NICOLÁS
NAVASA MAYO
Profesor Titular de Universidad
University of Massachusetts Amherst
Amherst Center, Estados UnidosPublications in collaboration with researchers from University of Massachusetts Amherst (9)
2015
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Ikaros mediates the DNA methylation-independent silencing of MCJ/DNAJC15 gene expression in macrophages
Scientific Reports, Vol. 5
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Serum C3 enhances complement receptor 3-mediated phagocytosis of Borrelia burgdorferi
International Journal of Biological Sciences
2013
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MCJ/DnaJC15, an endogenous mitochondrial repressor of the respiratory chain that controls metabolic alterations
Molecular and Cellular Biology, Vol. 33, Núm. 11, pp. 2302-2314
2012
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CD14 cooperates with complement receptor 3 to mediate MyD88-independent phagocytosis of Borrelia burgdorferi
Proceedings of the National Academy of Sciences of the United States of America, Vol. 109, Núm. 4, pp. 1228-1232
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Identification of synthetic host defense peptide mimics that exert dual antimicrobial and anti-inflammatory activities
Clinical and Vaccine Immunology, Vol. 19, Núm. 11, pp. 1784-1791
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Macrophage p38 mitogen-activated protein kinase activity regulates invariant natural killer T-cell responses during borrelia burgdorferi infection
Journal of Infectious Diseases, Vol. 206, Núm. 2, pp. 283-291
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Multiserotype protection elicited by a combinatorial prime-boost vaccination strategy against bluetongue virus
PLoS ONE, Vol. 7, Núm. 4
2011
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Growth temperature regulation of some genes that define the superficial capsular carbohydrate composition of Escherichia coli K92
FEMS Microbiology Letters, Vol. 320, Núm. 2, pp. 135-141
2010
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The tick saliva immunosuppressor, Salp15, contributes to Th17-induced pathology during Experimental Autoimmune Encephalomyelitis
Biochemical and Biophysical Research Communications, Vol. 402, Núm. 1, pp. 105-109