Estudio de casos clínicos de la forma nerviosa del maedivisna en la comunidad de Castilla y León

  1. J. Benavides 1
  2. N. Gómez 1
  3. M. Fuertes 1
  4. C. García Pariente 1
  5. J. González 1
  6. M. Ferreras 1
  7. J.F. García Marín 1
  8. V. Pérez 1
  1. 1 Universidad de León
    info

    Universidad de León

    León, España

    ROR https://ror.org/02tzt0b78

Book:
Producción Ovina y Caprina: XXIX Jornadas Científicas. VIII Internacionales de la Sociedad Española de Ovinotecnia y Caprinotecnia
  1. Fernando Forcada Miranda (coord.)
  2. Eduardo Angulo Asensio (coord.)
  3. José Antonio García de Jalón Ciércoles (coord.)
  4. Marcelo de las Heras Guillamón (coord.)
  5. Fernando López Gatius (coord.)
  6. Marianao Domingo Álvarez (coord.)
  7. Alfonso Abecia Martínez (coord.)

Publisher: Sociedad Española de Ovinotecnia y Caprinotecnia SEOC

Year of publication: 2004

Congress: Sociedad Española de Ovinotecnia y Caprinotecnia (SEOC). Jornadas (8. 2004. Lleida)

Type: Conference paper

Abstract

Maedi-Visna infection is widespread in Spain and the mammary and respiratory forms are the mostcommon. However, the nervous form or Visna would only appear sporadically. In this work, a clinical, lesionaland etiologic study is carried out. The importance of Visna among sheep showing neurological signs submittedto the Pathological Diagnostic Service of the Veterinary Faculty of Leon is also evaluated. Between 1995 and2003, a total of 3713 adult sheep were studied. From them, 682 showed nervous clinical symptoms. Seventy two(10,5%) of these animals had lesions on the central nervous system consistent with the nervous form of Maedi-Visna. The majority belonged to Assaf breed and were between 8 months and 9 years old. Tthey came fromdairy flocks managed in an intensive production system, located in Palencia, León, Zamora and Valladolidprovinces. PCR and immunohistochemistry techniques were employed for demonstration of the presence ofMaedi-Visna virus on lesions, while AGID and ELISA serological tests were also performed. The most commonclinical sign was hind limb ataxia, progressing to animal recumbence. Lesions, characterized by a nonsuppurativemeningoencephalitis, were mainly present at the brain stem and spinal cord white matter, althoughshowing different patterns.